Therapeutic vaccine candidates working with viral vectors encoding LMP2 have effectively induced virus-particular CD8⁺ T mobile responses in early-section scientific trials. Moreover, adoptive T cell therapies like posoleucel also goal LMP2 along with other latency proteins including EBNA1, reinforcing its importance as being a conserved and immunodominant antigen.
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EBNA-3a appears to control a subset of mobile genes, many of which overlap with All those regulated by EBNA2. The 2 proteins can act antagonistically or cooperatively to control gene expression. These findings position EBNA-3a as being a essential modulator of B mobile proliferation and survival for the duration of EBV latency, and as a relevant CD8⁺ T cell target in both of those standard immunological reports and translational research.
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